Ukuhlasela okukhulu kwenhliziyo - imiphumela, amathuba okuphila

I-infarction ye-myocardial yindlela elula yokungakwazi ukufakwa emisipha yenhliziyo yegazi ehlanganiswa ne-oxygen nezakhi. Lezi zifo ziholela eqinisweni lokuthi esikhathini esifushane, amangqamuzana ehliziyo kanye nezicubu ziyafa. Ngenxa yalokho, inhliziyo iyayeka. Kodwa ngisho nangesifo esikhulu senhliziyo, umuntu unethuba elikhulu lokuphila futhi, naphezu kwemiphumela, aphile impilo egcwele.

Yimaphi amathuba okusinda ngemuva kokuhlaselwa kwenhliziyo enkulu?

Ukuvuselelwa okusheshayo kwesiguli lapho kuqala ukuhlaselwa kwenhliziyo enkulu kunika ithuba lokusinda, kuvimbele ukuqala kwemiphumela emibi futhi kwenziwe ukuvuselelwa, okungenani kancane. Uma kungekho odokotela abaseduzane, ukuvuselelwa kufanele kwenziwe ngokwakho. Kumelwe:

  1. Qinisekisa ukuthi uhambo lwama-airway lubeka kanjani (ukubeka umuntu endaweni ephepheni, ukugoba ikhanda, ukukhipha izidumbu zangaphandle emlonyeni wakhe).
  2. Qinisekisa ukuthi isiguli siphefumula sisodwa.
  3. Qalisa umoya wokuphuza umoya ngaphandle kokuphefumula.

Ngokwe-pathology enjalo, umuntu angangena ku-coma (ngokushesha noma emahoreni ambalwa). Lokhu kubonisa ukulimala okujulile nokungaqondakali kwengqondo okubangelwa ukuphazamiseka kwemithambo yegazi. Uma isiguli sisehlakalweni se-coma ngemuva kokuhlaselwa kwenhliziyo okukhulu ngaphezu kwezinyanga ezingu-4, amathuba okuhlala ewela ngaphansi kwe-15%. Ukuqedela okuphelele kulokhu ngeke kwenzeke ku-100%.

Imiphumela yokuhlaselwa kwenhliziyo ejulile

Imiphumela ye-intensive heart attack is kakhulu. Emzimbeni izinqubo ezingaphenduki ziqala. Iningi labantu:

Imiphumela evamile ye-infarction enkulu ye-myocardial nayo i-heart aneurysm ne- thromboembolism . Kwezinye izimo, iziguli zithola i-pleurisy ne-edema ye-pulema. Ukuze uphazamisi olunzulu lwe-myocardial odongeni lwangaphakathi lwe-myocardium, imiphumela efana nokuhluleka kwenhliziyo nokushaqeka kwamagciwane kuyizici.

Izici zokuvuselelwa emva kokuphazamiseka

Ukuvuselelwa komuntu oye wahlaselwa yinhliziyo kuhloswe ukubuyisela umsebenzi womzimba kanye nempilo yengqondo. Ngokuqinisekile isiguli sidinga ukwelashwa ngokomzimba, ngokulawula ngokucophelela i-pulse. Lokhu kuzogcwalisa zonke izitho zomzimba ngegazi nezakhi. Ngaphezu kokuzivocavoca okukhethekile, isimiso senhliziyo sinemiphumela emihle ku:

Indima ebalulekile ekubuyiseleni komzimba idlala ngokudla. Ekudleni komuntu odinga ukuphulukisa imiphumela yokuhlaselwa kwenhliziyo ejulile, kumele kube nemikhiqizo esiza ukujwayelekile ukusebenza kwemisipha yenhliziyo. Lesi sinkwa, imifino eluhlaza nezithelo. Ukudla, okuyinto eyenza ukwakhiwa amaphilisi atherosclerotic, kufanele kukhishwe ekudleni. Ihlanganisa:

Ukuze ubuyele ekuphileni okuvamile emva kokuhlaselwa kwenhliziyo, kufanele uthathe imithi ehlukahlukene. Ngesikhathi sokuvuselelwa, zonke iziguli zibekwe izidakamizwa ezivimbela ukuthuthukiswa kwe-atherosclerosis kanye ne-thrombosis. Ezinye iziguli nazo zidinga ukusebenzisa i-beta-blockers ukwelashwa (i-Obsidan noma i-Anaprilin). Baqala ukusebenza ngendlela evamile ye-myocardium, ukuvimbela imiphumela yokukhathazeka ngokweqile nangokomzimba. Yamukela iminyaka eminingana, futhi ngezinye izikhathi kuze kube sekupheleni kokuphila. Ukuqeda ukwelashwa kwezidakamizwa kungabangela ukuphindaphinda, ukungena noma ezinye izinkinga.